Neuropathic pain after spinal cord injury differs fundamentally from nociceptive pain (normal pain from tissue damage). When the spinal cord is damaged, nerve pathways that transmit pain signals become disrupted and dysfunctional. The injured nervous system generates abnormal pain signals even without external stimuli, creating phantom sensations below the level of injury. This type of pain is classified into two main categories: at-level neuropathic pain (occurring at the site of spinal injury) and below-level neuropathic pain (occurring in areas below the injury site).
Medical research indicates that neuropathic pain typically develops within the first year after injury, though it can emerge months or even years later. The pain is often described as burning, shooting, stabbing, or electric-like sensations. Some patients experience allodynia (pain from normally non-painful stimuli like light touch) or hyperalgesia (exaggerated pain response). Unlike acute pain that serves a protective function, neuropathic pain serves no biological purpose and significantly impairs quality of life, sleep, mood, and rehabilitation progress.
When pursuing a [personal injury](/personal-injury) claim in California, documenting the onset, severity, and impact of neuropathic pain is crucial. This chronic condition substantially increases the value of spinal cord injury settlements because it represents ongoing suffering that may never fully resolve despite treatment.
- ✓At-level neuropathic pain affects 30-40% of SCI patients
- ✓Below-level neuropathic pain affects 30-50% of SCI patients
- ✓Pain intensity often rated 7-10 on pain scales
- ✓Condition may worsen over time without proper management