Syringomyelia is a chronic neurological condition characterized by the formation of a fluid-filled cavity, called a syrinx, within the spinal cord. In post-traumatic cases, this cyst typically develops at or near the site of the original spinal cord injury. The syrinx gradually expands over time, destroying nerve tissue and causing progressive neurological symptoms that may not appear until months or years after the initial trauma. Medical research indicates that between 1% and 4% of spinal cord injury patients will eventually develop post-traumatic syringomyelia, though some studies suggest the actual incidence may be higher due to underdiagnosis.
The exact mechanism by which syringomyelia develops after spinal trauma is not fully understood, but several theories exist. The most widely accepted explanation involves disruption of normal cerebrospinal fluid flow around the spinal cord, leading to fluid accumulation within the cord itself. Scar tissue formation, arachnoiditis (inflammation of the protective membranes around the spinal cord), and tethering of the spinal cord can all contribute to abnormal fluid dynamics. The syrinx typically begins as a small cavity but can extend both above and below the level of the original injury, sometimes spanning multiple spinal segments.
Symptoms of post-traumatic syringomyelia often develop insidiously and may include progressive weakness, ascending sensory loss (particularly loss of pain and temperature sensation), increased spasticity, worsening pain (often described as burning or aching), and loss of previously recovered function. Because these symptoms can be subtle initially and develop gradually, syringomyelia is sometimes not diagnosed until significant neurological deterioration has occurred. Advanced imaging with MRI is the gold standard for diagnosis, clearly showing the fluid-filled cavity within the spinal cord. Early detection and treatment are crucial for preventing further neurological damage and preserving remaining function.