Pressure sores develop when sustained pressure on the skin compresses blood vessels, depriving tissues of oxygen and nutrients. For spinal cord injury patients, several factors increase vulnerability to these wounds. Loss of sensation means patients cannot feel the discomfort that would normally prompt position changes. Reduced mobility prevents natural shifting of body weight. Muscle atrophy and loss of protective padding over bony prominences create pressure points. Additionally, impaired circulation below the injury level compromises the body's ability to heal damaged tissue.
The most common sites for pressure sores in SCI patients include the sacrum and coccyx (tailbone area), ischial tuberosities (sitting bones), heels, ankles, hips, and shoulder blades. These areas bear the most pressure during sitting or lying down. According to medical research, approximately 30-40% of spinal cord injury patients will develop at least one pressure ulcer during their lifetime, with higher rates among those with complete injuries and limited mobility. The National Spinal Cord Injury Statistical Center reports that pressure ulcers are among the most frequent complications requiring rehospitalization for SCI patients.
Pressure sores are classified into four stages based on severity. Stage 1 involves intact skin with non-blanchable redness. Stage 2 shows partial-thickness skin loss with exposed dermis. Stage 3 involves full-thickness skin loss with visible fat tissue. Stage 4, the most severe, involves full-thickness tissue loss with exposed muscle, tendon, or bone. Deep tissue injuries can also occur, where the surface may appear intact but underlying tissue is damaged. Understanding these classifications is crucial for catastrophic injury claims, as treatment costs and long-term impacts vary dramatically by stage.